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Atolife

Atolife is an add-on medication to diet to improve cholesterol levels. This medication is an alternative option to statins. Atolife is a cholesterol-lowering medication. It reduces the amount of cholesterol that is absorbed by the body, which helps lower total cholesterol and "bad" cholesterol (called LDL) in your blood.

Other names for this medication:
Atolife, Avascare, Etimibe, Ezedoc, Ezentia, Ezerem, Ezetib, Ezetimiba, Ezetrol, Ezevas, Ezibloc, Eziwa, Ezzicad, Filet, Imbibe, Lipezet, Liponorm, Orvas, Razel, Redustat, Storvas, Tonact, Visvas, Zeteze, Zetica, Zient, Zivast

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Also known as: Ezetimibe

Description

{name} (ezetimibe) reduces the level of total cholesterol and LDL (bad) cholesterol in the blood.

{name} is used to treat high cholesterol in people who cannot control their cholesterol levels by diet and exercise alone.

{name} works to reduce the amount of cholesterol your body absorbs. You should stay on a cholesterol-lowering diet while taking this medicine.

{name} is sometimes given together with other cholesterol-lowering medications.

Dosage

Usual Adult Dose of Atolife for Hyperlipidemia:

10 mg orally once a day Uses: -For use as monotherapy or in combination with an HMG-CoA reductase inhibitor (statin) as an adjunct to diet to reduce elevated total cholesterol (total-C), low-density lipoprotein cholesterol (LDL-C), apolipoprotein B (Apo B), and non-high-density lipoprotein cholesterol (non-HDL-C) in patients with primary (heterozygous familial and non-familial) hyperlipidemia -For use as combination therapy with fenofibrate as an adjunct to diet to reduce elevated total-C, LDL-C, Apo B, and non-HDL-C in patients with mixed hyperlipidemia -As an adjunct to diet to reduce elevated sitosterol and campesterol levels in patients with homozygous familial sitosterolemia -For use as combination therapy with atorvastatin or simvastatin to reduce elevated total-C and LDL-C levels in patients with homozygous familial hypercholesterolemia, as an adjunct to other lipid-lowering treatments (e.g., LDL apheresis) or if such treatments are unavailable

Usual Adult Dose of Atolife for Homozygous Familial Hypercholesterolemia:

10 mg orally once a day Uses: -For use as monotherapy or in combination with an HMG-CoA reductase inhibitor (statin) as an adjunct to diet to reduce elevated total cholesterol (total-C), low-density lipoprotein cholesterol (LDL-C), apolipoprotein B (Apo B), and non-high-density lipoprotein cholesterol (non-HDL-C) in patients with primary (heterozygous familial and non-familial) hyperlipidemia -For use as combination therapy with fenofibrate as an adjunct to diet to reduce elevated total-C, LDL-C, Apo B, and non-HDL-C in patients with mixed hyperlipidemia -As an adjunct to diet to reduce elevated sitosterol and campesterol levels in patients with homozygous familial sitosterolemia -For use as combination therapy with atorvastatin or simvastatin to reduce elevated total-C and LDL-C levels in patients with homozygous familial hypercholesterolemia, as an adjunct to other lipid-lowering treatments (e.g., LDL apheresis) or if such treatments are unavailable

Usual Adult Dose of Atolife for Sitosterolemia:

10 mg orally once a day Uses: -For use as monotherapy or in combination with an HMG-CoA reductase inhibitor (statin) as an adjunct to diet to reduce elevated total cholesterol (total-C), low-density lipoprotein cholesterol (LDL-C), apolipoprotein B (Apo B), and non-high-density lipoprotein cholesterol (non-HDL-C) in patients with primary (heterozygous familial and non-familial) hyperlipidemia -For use as combination therapy with fenofibrate as an adjunct to diet to reduce elevated total-C, LDL-C, Apo B, and non-HDL-C in patients with mixed hyperlipidemia -As an adjunct to diet to reduce elevated sitosterol and campesterol levels in patients with homozygous familial sitosterolemia -For use as combination therapy with atorvastatin or simvastatin to reduce elevated total-C and LDL-C levels in patients with homozygous familial hypercholesterolemia, as an adjunct to other lipid-lowering treatments (e.g., LDL apheresis) or if such treatments are unavailable

Usual Pediatric Dose for Hyperlipidemia:

10 years or older: 10 mg orally once a day Uses: -For use as monotherapy or in combination with a statin as an adjunct to diet to reduce elevated total-C, LDL-C, Apo B, and non-HDL-C in patients with primary (heterozygous familial and non-familial) hyperlipidemia -For use as combination therapy with a fenofibrate as an adjunct to diet to reduce elevated total-C, LDL-C, Apo B, and non-HDL-C in patients with mixed hyperlipidemia -As an adjunct to diet to reduce elevated sitosterol and campesterol levels in patients with homozygous familial sitosterolemia -For use as combination therapy with atorvastatin or simvastatin for the reduction of elevated total-C and LDL-C in patients with homozygous familial hypercholesterolemia, as an adjunct to other lipid-lowering treatments (e.g., LDL apheresis) or if such treatments are unavailable

Usual Pediatric Dose for Homozygous Familial Hypercholesterolemia:

10 years or older: 10 mg orally once a day Uses: -For use as monotherapy or in combination with a statin as an adjunct to diet to reduce elevated total-C, LDL-C, Apo B, and non-HDL-C in patients with primary (heterozygous familial and non-familial) hyperlipidemia -For use as combination therapy with a fenofibrate as an adjunct to diet to reduce elevated total-C, LDL-C, Apo B, and non-HDL-C in patients with mixed hyperlipidemia -As an adjunct to diet to reduce elevated sitosterol and campesterol levels in patients with homozygous familial sitosterolemia -For use as combination therapy with atorvastatin or simvastatin for the reduction of elevated total-C and LDL-C in patients with homozygous familial hypercholesterolemia, as an adjunct to other lipid-lowering treatments (e.g., LDL apheresis) or if such treatments are unavailable

Usual Pediatric Dose for Sitosterolemia:

10 years or older: 10 mg orally once a day Uses: -For use as monotherapy or in combination with a statin as an adjunct to diet to reduce elevated total-C, LDL-C, Apo B, and non-HDL-C in patients with primary (heterozygous familial and non-familial) hyperlipidemia -For use as combination therapy with a fenofibrate as an adjunct to diet to reduce elevated total-C, LDL-C, Apo B, and non-HDL-C in patients with mixed hyperlipidemia -As an adjunct to diet to reduce elevated sitosterol and campesterol levels in patients with homozygous familial sitosterolemia -For use as combination therapy with atorvastatin or simvastatin for the reduction of elevated total-C and LDL-C in patients with homozygous familial hypercholesterolemia, as an adjunct to other lipid-lowering treatments (e.g., LDL apheresis) or if such treatments are unavailable

Detailed Atolife dosage information

Overdose

Seek emergency medical attention or call the Poison Help line at 1-800-222-1222.

Storage

Store at room temperature between 15 and 30 degrees C (59 and 86 degrees F) away from moisture and heat. Keep container tightly closed. Throw away any unused medicine after the expiration date. Keep out of the reach of children.

Side effects

The most common side effects associated with Atolife are:

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Side effect occurrence does not only depend on medication you are taking, but also on your overall health and other factors.

Contraindications

You should not use Atolife if you are allergic to ezetimibe, or if you have:

moderate to severe liver disease.

Atolife is not approved for use by anyone younger than 10 years old.

You should not use Atolife with a "statin" cholesterol medicine (Zocor, Lipitor, Crestor, and others) if:

you have active liver disease; you are pregnant; or you are breastfeeding a baby.

Statin cholesterol medications can cause the breakdown of muscle tissue, which can lead to kidney failure. This happens more often in women, in older adults, or people who have kidney disease or poorly controlled hypothyroidism (underactive thyroid).

To make sure Atolife is safe for you, tell your doctor if you have:

liver disease; kidney disease; or unexplained muscle pain or weakness.

Tell your doctor if you are pregnant or breastfeeding.

Use effective birth control to prevent pregnancy while you are using Atolife with a statin medicine. Tell your doctor if you plan to become pregnant.

Do not breastfeed if you take Atolife with a statin medicine.

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However, the effects of subpressor doses of angiotensin II on the vasoconstrictor effects of norepinephrine have not been reported in humans. Biomarkers of thyroid function, genotoxicity and agricultural fungicide use. Details of pain severity, nausea, abdominal bloating and disruption to daily activities were recorded by means of clinician assessments and patient diaries. A 61-year-old woman with an abrupt onset of paralysis of the legs and impairment of the bladder and bowel function. Regulation of the human protein S gene promoter by liver enriched transcription factors.

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This is the first study to examine gastric emptying in an adequate number of healthy subjects by 13C breath tests. Absolute and relative risks were compiled using children of high/intermediate level salaried employees as the comparison group. Hepatosplenic lymphoma with a probable T-cell origin was diagnosed based on gross and histologic findings. An embolus, histologically identical with the aortic valve vegetation, was found in the left anterior descending artery at necropsy. These findings raise the possibility that NRG-induced activation of cdk5 works in part by recruiting PSD-95, a protein involved in regulating synaptic plasticity, to associate with ErbB4. We have obtained ribosomal DNA sequence information from several species in these groups. The SAP-1 B-box is part of a flexible linker of which 21 amino acids become ordered upon ternary complex formation. A 55-year- prochlorperazine drug class old woman who suffered from chronic diarrhea was diagnosed with lymphocytic colitis on colonic biopsy.

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This mutant has allowed synaptic electrophysiology after acute perturbation of a specific calcium-channel gene product, demonstrating that cac encodes a primary calcium channel functioning in neurotransmitter release. The 20-item Toronto Alexithymia Scale (TAS-20) measures three intercorrelated dimensions of alexithymia: (1) difficulties identifying feelings (DIF), (2) difficulties describing feelings (DDF), and (3) externally oriented thinking (EOT). The typical feature was a liquid echostructure in an endemic context. In the present manuscript we report that soluble PrPSc bound to and replicated within both GT1 neuronal cells and primary cortical neurons. Inducible nitric oxide synthase (iNOS) may be a critical factor in the repair of injured tissues. We found the method effective in approximately separating the contributions of these relaxation mechanisms, although the orientational-collision-induced interference makes a non-negligible contribution to the total response. Even in Holland there are moves to develop a hospice programme, to offer a radically alternative approach to terminal care. Six patients underwent endotracheal intubation, and in 5 of them long-term mechanical ventilation began at the age of 15 to 20 years.

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A 41-year-old caucasian woman without antecedents of interest was subjected to left catarct surgery involving phacoemulsification with capsular sac implantation of a new type of multifocal lens. Rabbit eyes were exposed to sulfur mustard (HD) vapor (370, 420 microg/l) for a period of two minutes. There may be an advantage to administration of PB over PA, since the administration of PB results in significant exposure to both active compounds. Quantification of carcinoembryonic antigen (CEA) mRNA in peritoneal washes by real-time reverse-transcriptase polymerase chain reaction (RT-PCR) was found to be a more sensitive method to detect free cancer cells. Among the exogenous proteins initiation factor eIF2 is the best substrate and is 7.8 times more efficiently phosphorylated than casein. Trials exploring tenormin 100 mg tablet controlled, safe intra-patient dose escalation with the intent of achieving grade 3 or 4 neutropenia in patients with SCLC are warranted. Adsorption patterns of pellicles formed in a flow cell system under a moderate shear rate of 21 s-1, were studied with a replica technique. Minute virus of mice nonstructural protein NS1 is a multifunctional protein that is involved in many processes necessary for virus propagation. Preliminary results of two nation-wide epidemiological studies of drug use in Greece: a study of known cases and a general population survey. Cross-sectional telephone survey with participants randomly selected from the electronic NSW telephone directory.

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The inhibitory reactions of the gastroduodenal zone during electrical stimulation of the area of the afferent innervation by the vagus nerve on the human external ear. The albumin loss in ultrafiltrate by Sureflux 150E was greater than AV-400. For the time being it is not possible to give a satisfactory explanation of the decrease in the ECOD activity of the hepatoses. Because sodium citrate is known to increase both gastric pH and gastric volume while metoclopramide decreases gastric volume, we evaluated, in a double-blind randomized study, the effect of combining metoclopramide with sodium citrate on gastric pH and volume after induction of anesthesia in 60 female patients to determine whether gastric pH could be increased at the same time that gastric volume was decreased. Recently it was demonstrated that beta-actin can be produced in Saccharomyces cerevisiae by using the expression plasmid pY beta actin (R. Conditioned medium could be successfully used to replace feeder cells in NG2 stock cultures.

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Of the 52 patients, 44 had a static gantry dynamic multileaf collimated (SG-DMLC) IMRT treatment plan, seven of which had quality assurance (QA) measurements. Non-drinkers have elevated levels of psychological distress but a recent study reported no elevation in prevalence of diagnosed disorders. In summary, spatial and temporal analysis of terminal organelle formation has yielded insights into the nature of M. During the past two years, floating stones within the gallbladder have been demonstrated in five patients, by both oral cholecystography and sonography. It is concluded that predictable changes in pain behaviors occur within infancy and toddlerhood. This is the first reported case of a gynecologic cancer in which the paraneoplastic syndrome has been conclusively shown by immunohistochemical analysis to be due to ectopic parathormone. Cell-TakTM-treated plates provided the best environment for isolation and purification of this cell type as judged by lack of fibroblast contamination, growth rate, and cell morphology. Follicular thyroid cancer treated at the Mayo Clinic, 1946 through 1970: initial manifestations, pathologic findings, therapy, and outcome.

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