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Amlokos (Atenolol)
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Amlokos

Amlokos is a selective beta blocker. It works by blocking specific (natural) compounds that may bind to receptors on the heart. This slows down your heart rate so your heart doesn't have to work as hard.

Other names for this medication:
Adipin, Amdinx, Amlica, Amloheart, Amlonol, Amnol, Amsten, Amtas, Aniloc, Atamo, Atemarc, Atemide, Atormin, Atzee, Avino, Bardateno, Betatrop, Cabeta, Calock, Cardenol, Cardica, Celbeta, Etotan, Loscard, Myamlo, Myodura, Nifetolol, Ortibid, Otenol, Teenol, Tefifin, Tenocheck, Tenoclor, Tensicard, Tensimin, Tentoric

Similar Products:
Atarax, Lamictal, Nexium, Neoral, Vasotec, Aceon, Arava, Intagra, Fosamax, Provigil

Also known as: Atenolol

Description

{name} (atenolol) is a beta-blocker that affects the heart and circulation (blood flow through arteries and veins).

{name} is used to treat angina (chest pain) and hypertension (high blood pressure).

{name} is also used to lower the risk of death after a heart attack.

Dosage

A nurse or other trained health professional will give you this medicine. This medicine is given through a needle placed in one of your veins .

Detailed Amlokos dosage information

Overdose

Seek emergency medical attention or call the Poison Help line at 1-800-222-1222.

Overdose symptoms may include extreme weakness or lack of energy, very slow heart rate, shortness of breath, or fainting.

Storage

Store at room temperature between 20 and 25 degrees C (68 and 77 degrees F) away from moisture and heat. Keep container tightly closed. Throw away any unused medicine after the expiration date. Keep out of the reach of children.

Side effects

The most common side effects associated with Amlokos are:

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Side effect occurrence does not only depend on medication you are taking, but also on your overall health and other factors.

Contraindications

You should not use Amlokos if you are allergic to atenolol, or if you have:

a serious heart condition such as "AV block" (second or third degree); very slow heartbeats; or heart failure.

To make sure Amlokos is safe for you, tell your doctor if you have:

congestive heart failure; coronary artery disease (hardened arteries); asthma, bronchitis, emphysema; diabetes; overactive thyroid; liver or kidney disease; pheochromocytoma (tumor of the adrenal gland); peripheral vascular disease such as Raynaud's syndrome; or allergies (or if you are undergoing allergy treatments or skin-testing).

Using Amlokos during pregnancy could harm the unborn baby. Tell your doctor if you are pregnant or if you become pregnant while using this medicine.

Atenolol can pass into breast milk and may harm a nursing baby. Tell your doctor if you are breast-feeding a baby.

Amlokos is not approved for use by anyone younger than 18 years old.

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Operational feasibility of using loop-mediated isothermal amplification for diagnosis of pulmonary tuberculosis in microscopy centers of developing countries. Tobacco industry cigarette advertising is associated with increased adolescent smoking, while counter tobacco advertising is associated with reduced smoking. Twelve Lewis rats received a local perianal injection of Bioplastique implants. pollen in China (Liliaceae): taxonomic and phylogenetic implications and pollen evolution related to environmental conditions. A significant decrease in testis and epididymis weight was however observed following high doses of gossypol. Strong consideration should be given for foregoing blood culture in a non-toxic child 3-36 months of age with FWS. Similar to patients with mutations in NDP, LRP5, or FZD4, the phenotypes associated with TSPAN12 mutations showed great variations between different individuals within a family and between the two eyes in individual patients. To evaluate the pregnancy results of an ovarian hyperstimulation protocol for IVF-ET that combines GnRH agonist down-regulation, cessation of GnRH agonist therapy with the onset of menstruation, and high-dose gonadotropin administration in low responders. Peak heart rate (PHR-II) also remained significantly slower (P less than 0.05).

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To describe parathyroid hormone-related protein (PTHrP) as a mediator of hypercalcemia in sarcoidosis. Minimization of chronic plasma viremia in rhesus macaques immunized with synthetic HIV-1 Tat peptides and infected with a chimeric simian/human immunodeficiency virus (SHIV33). A quantitative structure-activity relationship (QSAR) study has been made on a series of piperidine sulfonamide aryl hydroxamic acid analogs acting as matrix metalloproteinase (MMP) inhibitors. Limited information on epidemiologic patterns of KSHV, with none focusing on heterosexual transmission, is available in mainland China. Further, results from this study indicate that the role of religiosity, found in this study to predict both financial and health care planning, warrants further exploration. To report the utilization of ANC services by women living in tribal and rural areas in the district of Visakhapatnam, Andhra Pradesh, India.

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Nerve conduction velocity and axonal transport of 6-phosphofructokinase activity in galactose-fed rats. We provide dynamic surface tension data of serum and cerebrospinal fluid that were collected from healthy volunteers and patients with rheumatic, neurological or oncological diseases. High-throughput genome-wide methods have been pivotal in driving the latest progress in the field. This article reports the water repellency mechanism of water strider legs. Some crustacean axons remain functional for months after injury. Antibiotic resistance to CAM was analyzed by molecular and culture methods.

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This study demonstrates the occurrence of a focal dysfunction of cerebral perfusion in children with cerebellar mutism after posterior fossa surgery. The purpose of this study is to investigate functional changes of Th17/Th1, Th17, Th1, Th2 and Treg cells in ACS patients. Competition between actin-WCA and glia maturation factor (GMF) for binding to Arp2/3 complex suggests that during activation the first actin monomer binds at the barbed end of Arp2. They are also a medication of choice for angina after an infarction. Suppression of Fn14 expression in C2C12 myoblasts also inhibited the differentiation-associated increase in the activity of serum response factor and RhoA GTPase.

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