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Adaple (Adapalene)
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Adaple

Adaple treats acne deep in the pores at the source, prevents new acne from forming and, as a result of reduced acne, restores your skin’s natural texture and tone. Adaple has been prescribed to more than 40 million people over the last 20 years.

Other names for this medication:
Aclene, Acneril, Acneris, Acniriv, Acnovate, Adaferin, Adapalene, Adapen, Adaple, Adaret, Adgraze, Adiff, Adina, Aditop, Adlene, Alangel, Apalene, Apgel, Clinred, Daple, Deriva, Difra, Edplin, Faceclin, Labopene, Medapine, Rozac, Soflene, Vandep, Zolvee

Similar Products:
Prilosec, Epivir, Geodon, Cardizem, Differin

Also known as: Adapalene

Description

Adaple is a third-generation topical retinoid primarily used in the treatment of acne. Adaple is a gel used for the treatment of acne (blackheads, whiteheads and pimples) of the face, chest or back. Adaple gel is a clear gel that powers deep inside your pores to control the buildup that leads to acne. It helps prevent the formation of new acne lesions, and can also help get rid of lesions already present. Adaple works under the skin surface to unclog pores and clear up pimples. Adaple is also known as Adapalene, Adapen, Adaferin. Generic name of Adaple is Adapalene.

Dosage

Follow the directions for using this medicine provided by your doctor. Use Adaple exactly as directed. Adaple is best applied after washing and drying your skin. Apply a thin layer of gel to the affected areas once daily at bedtime. Avoid the eyes, lips and nose. Do not allow this preparation to come into contact with the mucous membranes (e.g. mouth, vagina, inside of nose etc.). Try not to touch the treated areas overnight. Do not use any harsh soaps or exfoliants while you are being treated with Adaple. Adaple should not be used on broken, sunburnt or infected skin. You may notice a slight change of colour of the skin you are applying this preparation to. This is only temporary and should return to normal soon after finishing treatment with Adaple. Continue using Adaple until no new acne spots appear. You may need to use it for several months. You should notice some clinical improvement after 4-8 weeks and more improvement after further use. There may be a worsening of acne during the first few weeks of therapy because Adaple promotes the growth of pimples that have begun to form but are not yet visible. Therefore, treatment should not be stopped if acne appears to be worsening. Use a sunscreen cream and wear protective clothing if exposure to strong or prolonged sunlight cannot be avoided. Weather extremes such as wind or cold may also be irritating.

Overdose

Seek emergency medical attention if you think you have used too much of this medicine. Overdose symptoms may include skin redness, scaling, or irritation.

Storage

Store the medicine in a closed container at room temperature, away from heat, moisture, and direct light. Keep from freezing.

Keep out of the reach of children.

Do not keep outdated medicine or medicine no longer needed.

Detailed Adaple dosage information

Side effects

The most common side effects associated with Adaple are:

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Side effect occurrence does not only depend on medication you are taking, but also on your overall health and other factors.

Contraindications

You should not use Adaple if you are allergic to adapalene.

Ask a doctor or pharmacist if it is safe for you to use this medicine if you have other medical conditions.

It is not known whether adapalene passes into breast milk or if it could harm a nursing baby. Do not use this medicine without a doctor's advice if you are breast-feeding a baby.

It is not known whether Adaple will harm an unborn baby. Do not use this medicine without a doctor's advice if you are pregnant.

Adaple is not approved for use by anyone younger than 12 years old.

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ACE inhibition reduces left ventricular mass independent of pressure without affecting coronary flow and flow reserve in spontaneously hypertensive rats. However, some studies indicate that steroid action can be extremely rapid and therefore unlikely to be through a genomic mechanism. Studies of proteinaceous cysteine protease inhibitors originated with the discovery of cystatins in the 1960s. Fluorescence light microscopy revealed the localization of QD705 in tissues. With highly asymmetric involvement, neonatal familial exudative vitreoretinopathy can mimic persistent hyperplastic primary vitreous. The rich information contained in these profiles, combined with developments in data analysis, has stimulated research and development to apply the powder technique to microcrystalline protein samples. The active peptide fraction can be extracted in high pH buffer from calf thymus native chromatin previously deproteinized by chloroform-isamyl alcohol and phenol. The diagnosis of hydatidosis was confirmed in one patient with discordant results (IFI/ES) and eliminated for the 3 remaining patients, in which these data were clinically confirmed later on. Provided that the recommended guidelines for metformin use are strictly adhered to, its widespread use would be safe and the incidence of MALA will be further reduced.

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Serum P I NP, in contrast to serum bone alkaline phosphatase (BAP) and intact osteocalcin (OC), did not show any significant change between before and after a hemodialysis session. Blood ketone bodies, bicarbonates, lactic acid and glucose before and after standard and all-out exercise in well trained and untrained adolescents. Irregular current spikes follow after the augmentation of the current induced when the phase-boundary potential is brought close to the mid-point potential of the transferring surfactant ions. We describe the case of a renal transplant patient who developed a disseminated HSV-2 infection with repeated urinary extravasations. Full-length cDNAs specific for oligodendrocyte- or astrocyte-specific genes were labeled with digoxigenin using the random primer method. We also studied the effect of prolonged capsaicin treatment which should predominantly impair the function of nociceptive C-fibers. Its pathogenesis remains unclear even if the initial injury likely occurs in the endothelial cells of central veins.

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Rats easily discriminate between two types of subcortical brain shock which differ in reinforcing properties. Ionizing radiation has a detrimental effect on the human body, particularly in children. The gel chromatographic behavior in 6M guanidium chloride of T-H glycopolypeptide with disulfide bonds intact as compared with its gel chromatographic behavior with disulfide bonds broken indicated that the glycopolypeptide is highly constrained by intrachain disulfide bonds. Dosage was 20 mg clomipramine during the first three days of treatment, increasing gradually over the next few weeks to a maximum daily dose of 200 mg clomipramine unless side-effects dictated a lower dose. The percentage of viable cells was determined by the 3-(4,5-dimethylthiazol-2-yl)-2,5-diphenyl tetrazorium bromide (MTT) assay. The synergistic effect of TLR2 and NOD2 ligands on RAC activation might be explained by the observations that bacterial lipoprotein (BLP, a TLR2 ligand) was able to upregulate NOD2 expression and the combination of BLP and muramyldipeptide (MDP, a NOD2 ligand) enhanced the expression of RICK (Rip2), the signaling molecule of NOD2. Blastocyst attachment to the lining of the mammalian uterus during early implantation involves the initial apposition of the trophoblast to the uterine epithelial surface. Loss of p16INK4a results in increased glucocorticoid receptor activity during fibrosarcoma development.

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